{"id":6829,"date":"2024-04-10T01:59:41","date_gmt":"2024-04-10T01:59:41","guid":{"rendered":"https:\/\/longchangchemical.com\/?post_type=product&p=6829"},"modified":"2025-12-17T02:18:24","modified_gmt":"2025-12-17T02:18:24","slug":"beta-nicotinamide-mononucleotide-cas-1094-61-7","status":"publish","type":"product","link":"https:\/\/longchangchemical.com\/tr\/product\/beta-nicotinamide-mononucleotide-cas-1094-61-7\/","title":{"rendered":"beta-nikotinamid monon\u00fckleotid CAS 1094-61-7"},"content":{"rendered":"

Beta-Nikotinamid Monon\u00fckleotidh\u0131zl\u0131 Ayr\u0131nt\u0131lar<\/strong><\/p>\n

Kimyasal Ad\u0131: Beta-Nikotinamid Monon\u00fckleotid<\/p>\n

CAS No: 1094-61-7<\/p>\n

Molek\u00fcler Form\u00fcl: C11H15N2O8P<\/p>\n

Beta-Nikotinamid Monon\u00fckleotidYap\u0131:\"Beta-Nikotinamid<\/p>\n

Molek\u00fcl a\u011f\u0131rl\u0131\u011f\u0131: 334,22<\/p>\n

G\u00f6r\u00fcn\u00fc\u015f: Beyaz toz<\/p>\n

Assay:97%~100.0%<\/p>\n

Beta-Nikotinamid Monon\u00fckleotid Tipik \u00d6zellikler<\/strong><\/p>\n\n\n\n\n\n\n\n\n\n
\u00d6\u011fe<\/td>\nTeknik \u00d6zellikler<\/td>\n<\/tr>\n
G\u00f6r\u00fcn\u00fc\u015f<\/td>\nBeyaz toz<\/td>\n<\/tr>\n
\u00c7\u00f6zeltinin g\u00f6r\u00fcn\u00fcm\u00fc (5% w\/v su i\u00e7inde)<\/td>\nRenksiz ila soluk sar\u0131 ve berrak<\/td>\n<\/tr>\n
Su<\/td>\n\u22646%<\/td>\n<\/tr>\n
Arsenik<\/td>\n\u22641 ppm<\/td>\n<\/tr>\n
A\u011f\u0131r metaller<\/td>\n\u226410 ppm<\/td>\n<\/tr>\n
Tahlil<\/td>\n97%~100.0%<\/td>\n<\/tr>\n<\/tbody>\n<\/table>\n

Beta-Nikotinamid Monon\u00fckleotidKullan\u0131m<\/strong><\/p>\n

Ya\u015flanma Kar\u015f\u0131t\u0131 Ara\u015ft\u0131rmalar, Ya\u015flanma Kar\u015f\u0131t\u0131 Sa\u011fl\u0131k \u00dcr\u00fcnleri Hammaddeleri<\/p>\n

Beta-Nikotinamid Monon\u00fckleotidPaketleme ve Nakliye<\/strong><\/p>\n

Ambalaj: 25kg \/ Karton kova<\/p>\n

Teslimat: Tehlikeli olmayan kargo<\/p>\n

Beta-Nikotinamid Monon\u00fckleotidDepolama<\/strong><\/p>\n

Do\u011frudan g\u00fcne\u015f \u0131\u015f\u0131\u011f\u0131ndan ka\u00e7\u0131nmak i\u00e7in kuru, havaland\u0131r\u0131lm\u0131\u015f alanlarda saklay\u0131n.<\/p>\n

\u015eimdi Bize Ula\u015f\u0131n!<\/b><\/strong><\/h2>\n

Fiyata ihtiyac\u0131n\u0131z varsa, l\u00fctfen a\u015fa\u011f\u0131daki forma ileti\u015fim bilgilerinizi doldurun, genellikle 24 saat i\u00e7inde sizinle ileti\u015fime ge\u00e7ece\u011fiz. Bana e-posta da g\u00f6nderebilirsiniz info@longchangchemical.com<\/span><\/a>\u00a0\u00c7al\u0131\u015fma saatleri i\u00e7inde (8:30 - 6:00 UTC+8 Pzt.~Sat.) veya h\u0131zl\u0131 yan\u0131t almak i\u00e7in web sitesi canl\u0131 sohbetini kullan\u0131n.<\/b><\/strong><\/h4>\n
\n

\u0130skemik serebrovask\u00fcler hastal\u0131k, ge\u00e7ici serebral iskemi ve serebral enfarkt\u00fcs olmak \u00fczere iki t\u00fcr i\u00e7erir. Bunlar aras\u0131nda serebral enfarkt\u00fcs, \u00c7in t\u0131bb\u0131 te\u015fhisinde iskemi inmesi (IS) olarak adland\u0131r\u0131l\u0131r ve beyin dokusu b\u00f6lgelerindeki lokal kan ak\u0131m\u0131 engellerinin \u00e7e\u015fitli nedenlerine ba\u011fl\u0131 olarak beyin dokusunun iskemik ve hipoksik nekrozu ile sonu\u00e7lanan bir dizi n\u00f6rolojik disfonksiyondur. \u00d6l\u00fcmc\u00fcll\u00fc\u011f\u00fc ve sakatl\u0131k oran\u0131 y\u00fcksektir ve patolojik mekanizmas\u0131 karma\u015f\u0131kt\u0131r, her y\u0131l d\u00fcnyaya b\u00fcy\u00fck bir ekonomik y\u00fck getirmektedir. Oksijen glukoz yoksunlu\u011fu (OGD), IS'nin en yayg\u0131n kullan\u0131lan in vitro modelidir ve temel ve preklinik inme ara\u015ft\u0131rmalar\u0131nda yayg\u0131n olarak kullan\u0131lmaktad\u0131r.<\/p>\n

H\u00fccresel otofaji (autophagy) \u00f6karyotik h\u00fccrelerde yayg\u0131n olarak bulunur ve h\u00fccre sa\u011fkal\u0131m\u0131n\u0131n ve \u00f6l\u00fcm\u00fcn\u00fcn d\u00fczenlenmesinde \u00f6nemli bir rol oynar. \u00c7ok say\u0131da \u00e7al\u0131\u015fma otofajinin IS'nin olu\u015fumu ve geli\u015fimi ile yak\u0131ndan ili\u015fkili oldu\u011funu \u00f6ne s\u00fcrm\u00fc\u015ft\u00fcr. Baz\u0131 \u00e7al\u0131\u015fmalar n\u00f6ronal otofajinin iskemik beyin hasar\u0131n\u0131 hafifletti\u011fini g\u00f6sterirken, di\u011ferleri n\u00f6ronal otofajinin iskemik beyin hasar\u0131n\u0131 \u015fiddetlendirdi\u011fini bildirmi\u015ftir, ancak spesifik mekanizma net de\u011fildir.
\n\u03b2-nikotinamid monon\u00fckleotid (NMN) \u00e7ok \u00e7e\u015fitli g\u0131dalarda bulunur ve h\u00fccresel ya\u015flanman\u0131n d\u00fczenlenmesi ve organizman\u0131n normal i\u015flevinin s\u00fcrd\u00fcr\u00fclmesi i\u00e7in gereklidir ve bir\u00e7ok \u00f6nemli h\u00fccre i\u00e7i sinyal yolunun iletiminde rol oynar. NMN'nin in vitro uygulamas\u0131n\u0131n NAD+'yi h\u0131zla NAD+'ye d\u00f6n\u00fc\u015ft\u00fcrd\u00fc\u011f\u00fc ve h\u00fccresel ya\u015flanmay\u0131 d\u00fczenledi\u011fi ve normal v\u00fccut fonksiyonlar\u0131n\u0131 korudu\u011fu bildirilmi\u015ftir.NMN, IS'nin neden oldu\u011fu NAD+ azalmas\u0131n\u0131 telafi edebilir ve serebral iskemik n\u00f6ronlar\u0131n hasar\u0131n\u0131 iyile\u015ftirebilir. Bu, NMN'nin IS'de bir rol oynad\u0131\u011f\u0131n\u0131 d\u00fc\u015f\u00fcnd\u00fcrmektedir. Baz\u0131 \u00e7al\u0131\u015fmalar NMN'nin n\u00f6rovask\u00fcler rejenerasyonu te\u015fvik edebilece\u011fini, serebral mikrovask\u00fcler endotel fonksiyonunu, anti-enflamasyonu ve anti-apoptozu iyile\u015ftirebilece\u011fini g\u00f6stermi\u015ftir. Otofajiyi d\u00fczenleyerek iskemik beyin hasar\u0131 kar\u015f\u0131t\u0131 bir rol oynayabilece\u011fini \u00f6ne s\u00fcren birka\u00e7 \u00e7al\u0131\u015fma da vard\u0131r, ancak NMN'nin IS'ye kar\u015f\u0131 otofajiyi d\u00fczenledi\u011fi spesifik mekanizma net de\u011fildir.<\/p>\n

Bu \u00e7al\u0131\u015fman\u0131n amac\u0131, bir oksijen \u015feker yoksunlu\u011fu PC12 h\u00fccre modeli olu\u015fturmak ve in vitro MTT ile h\u00fccre sa\u011fkal\u0131m oran\u0131n\u0131 \u00f6l\u00e7mek, otofagozomlar\u0131 ve otofagolizozomlar\u0131 transmisyon elektron mikroskobu ile tespit etmektir, MDC ile otofagozomlar\u0131n floresan yo\u011funlu\u011funu g\u00f6zlemlemek ve NMN'nin PC12 h\u00fccrelerinde OGD kaynakl\u0131 otofaji hasar\u0131 \u00fczerindeki etkisini a\u00e7\u0131kl\u0131\u011fa kavu\u015fturmak i\u00e7in Western blot ile LC3-II\/LC3-I, Beclin1, p62 ve P-mTOR\/mTOR otofaji ile ili\u015fkili proteinlerin ekspresyonunu tespit etmek.<\/p>\n

2017 Lancet \u00e7al\u0131\u015fmas\u0131, inmenin h\u0131zl\u0131 ilerleme, y\u00fcksek \u00f6l\u00fcmc\u00fcll\u00fck ve sakatl\u0131k ile \u00c7in'de y\u0131llarca ya\u015fam kayb\u0131na neden olan ba\u015fl\u0131ca hastal\u0131k oldu\u011funu g\u00f6stermi\u015ftir. Bunlardan \u0130S, t\u00fcm inmelerin y\u00fczde 60 ila 70'ini olu\u015fturmaktad\u0131r ve \u0130S, bir kan damar\u0131ndaki t\u0131kan\u0131kl\u0131k nedeniyle beynin bir b\u00f6lgesine kan ak\u0131\u015f\u0131n\u0131n azalmas\u0131ndan kaynaklanmaktad\u0131r. Beyin dokusundaki iskemi ve hipoksi, h\u00fccre otofajisi, apoptoz, oksidatif stres, h\u00fccre i\u00e7i kalsiyum a\u015f\u0131r\u0131 y\u00fcklenmesi gibi bir dizi patofizyolojik yan\u0131t\u0131 tetikler. IS'den kurtulanlar\u0131n iyile\u015fme s\u00fcreci uzun s\u00fcrmekte ve bu da k\u00fcresel ekonomik y\u00fck\u00fc art\u0131rmaktad\u0131r. \u015eu anda piyasada \u0130D'ye kar\u015f\u0131 kullan\u0131lan ila\u00e7lar i\u00e7in bir\u00e7ok kontrendikasyon bulunmaktad\u0131r. Bu nedenle, \u0130D'yi \u00f6nlemek ve tedavi etmek i\u00e7in s\u00fcrekli olarak ila\u00e7lar\u0131n ara\u015ft\u0131r\u0131lmas\u0131 gerekmektedir. OGD, IS'nin in vitro \u00e7al\u0131\u015fmas\u0131 i\u00e7in klasik bir modeldir ve PC12 h\u00fccre hatt\u0131, iskemi-hipoksi hasar\u0131 \u00e7al\u0131\u015fmas\u0131nda s\u0131kl\u0131kla kullan\u0131lan n\u00f6ronal hasar\u0131 incelemek i\u00e7in en yayg\u0131n kullan\u0131lan h\u00fccre hatlar\u0131ndan biridir. Bu nedenle, bu deneyde in vitro \u00e7al\u0131\u015fma i\u00e7in OGD PC12 h\u00fccre modeli kullan\u0131lm\u0131\u015ft\u0131r.<\/p>\n

NMN, insan v\u00fccudunda ve bir\u00e7ok g\u0131dada do\u011fal olarak bulunan, molek\u00fcl a\u011f\u0131rl\u0131\u011f\u0131 334,22 olan bir maddedir. Nikotinamid-fosfat riboziltransferaz reaksiyonunun bir \u00fcr\u00fcn\u00fcd\u00fcr ve NAD+'nin temel \u00f6nc\u00fclerinden biridir. Baz\u0131 \u00e7al\u0131\u015fmalar, organizmalardaki NMN seviyesini d\u00fczenleyerek, kardiyovask\u00fcler ve serebrovask\u00fcler hastal\u0131klar, n\u00f6rodejeneratif hastal\u0131klar ve ya\u015flanman\u0131n dejeneratif hastal\u0131klar\u0131 \u00fczerinde daha iyi bir terap\u00f6tik ve onar\u0131c\u0131 etkiye sahip oldu\u011funu bulmu\u015ftur. NMN uygulamas\u0131n\u0131n s\u0131\u00e7an MCAO modelinde enfarkt\u00fcs alan\u0131n\u0131 ve n\u00f6rolojik fonksiyon hasar\u0131n\u0131 azaltabilece\u011fi de bildirilmi\u015ftir. Bu deneyde, MTT ile OGD'nin PC12 h\u00fccrelerinin hayatta kalma oran\u0131n\u0131 azaltabilece\u011fi ve 400, 800 ve 1600 \u03bcmol\/L konsantrasyonlar\u0131ndaki NMN'nin OGD PC12 h\u00fccrelerinin hayatta kalma oran\u0131n\u0131 art\u0131rd\u0131\u011f\u0131, en y\u00fcksek hayatta kalma oran\u0131n\u0131n 800 \u03bcmol\/L konsantrasyonunda oldu\u011fu g\u00f6sterilmi\u015ftir; oysa, 200 ve 3200 \u03bcmol\/L NMN konsantrasyonlar\u0131nda h\u00fccre hayatta kalma oran\u0131nda \u00f6nemli bir fark yoktu, bu da 200 \u03bcmol\/L NMN konsantrasyonunun PC12 h\u00fccrelerinin hayatta kalma oran\u0131n\u0131 art\u0131rabilece\u011fini g\u00f6stermektedir. Bu, 200 \u03bcmol\/L konsantrasyonun OGD ile ind\u00fcklenen PC12 h\u00fccrelerinin hayatta kalma oran\u0131n\u0131 art\u0131rmak i\u00e7in hen\u00fcz etkili bir konsantrasyon olmad\u0131\u011f\u0131n\u0131, 3200 \u03bcmol\/L konsantrasyonun ise \u00e7ok y\u00fcksek olabilece\u011fini ve h\u00fccrelerin kendilerine bir miktar zarar verebilece\u011fini g\u00f6stermektedir.<\/p>\n

Tip II programl\u0131 h\u00fccre \u00f6l\u00fcm\u00fc olarak bilinen h\u00fccresel otofaji, h\u00fccrelerin hayatta kalmalar\u0131n\u0131, farkl\u0131la\u015fmalar\u0131n\u0131, b\u00fcy\u00fcmelerini ve stabilitelerini s\u00fcrd\u00fcrmek i\u00e7in otofaji ile ili\u015fkili genlerin d\u00fczenlenmesi alt\u0131nda kendi hasarl\u0131, denat\u00fcre veya ya\u015flanm\u0131\u015f makromolek\u00fcllerinin yan\u0131 s\u0131ra organellerini bozmak i\u00e7in lizozomlar\u0131 kulland\u0131klar\u0131 bir s\u00fcre\u00e7tir. \u00c7al\u0131\u015fma tablosu, otofajinin IS'den sonra ind\u00fcklendi\u011fini ve ayn\u0131 zamanda otofajinin IS patolojik s\u00fcrecinin geli\u015fimine de e\u015flik etti\u011fini ve IS'nin d\u00f6rt evresinde, yani akut, subakut, iyile\u015fme ve sekel evrelerinde farkl\u0131 d\u00fczenleyici roller oynad\u0131\u011f\u0131n\u0131 g\u00f6stermektedir.<\/p>\n

mTOR, 289 kDa nispi molek\u00fcler k\u00fctleye sahip atipik bir serin \/ treonin protein kinazd\u0131r. farkl\u0131 proteinleri, mTORCl ve mTORC2 olmak \u00fczere 2 farkl\u0131 kompleks olu\u015fturmak i\u00e7in ba\u011flan\u0131r. mTORCl, rapamisine duyarl\u0131d\u0131r ve b\u00fcy\u00fcme fakt\u00f6rlerinin ve besin sinyallerinin entegrasyonundan sorumludur ve esas olarak h\u00fccresel otofajiyi, ribozom biyogenezini, protein translasyonunu ve lipid sentezini d\u00fczenler. mTORT, otofajinin valfi olarak kabul edilir. \u00c7al\u0131\u015fmalar, fosforile mTOR'un oksijen \u015feker yoksunlu\u011fu hasar\u0131n\u0131 hafifletebilece\u011fini ve h\u00fccrelerin korunmas\u0131nda rol oynayabilece\u011fini g\u00f6stermi\u015ftir. 3-MA yayg\u0131n olarak kullan\u0131lan bir otofaji inhibit\u00f6r\u00fcd\u00fcr. rAPA, mTORC1'i inhibe ederek otofajinin ba\u015flamas\u0131n\u0131 ind\u00fckleyen bir mTOR inhibit\u00f6r\u00fcd\u00fcr ve ayn\u0131 zamanda bir otofaji aktivat\u00f6r\u00fc olarak da adland\u0131r\u0131l\u0131r. Bu deneyde, 3-MA ve RAPA grubu PC12 h\u00fccrelerinde OGD ile ind\u00fcklenen otofajinin d\u00fczenlenmesine m\u00fcdahale etmek i\u00e7in ve 3-MA, RAPA ve ila\u00e7 kombinasyonu grubu da NMN'nin PC12 h\u00fccrelerinde OGD ile ind\u00fcklenen otofaji \u00fczerinde 3-MA veya RAPA'n\u0131n etkilerine kar\u015f\u0131 koyup koyamayaca\u011f\u0131n\u0131 g\u00f6zlemlemek i\u00e7in kurulmu\u015ftur.<\/p>\n

Beclin-1, LC3 ve p62 proteinlerinin seviyeleri, otofajinin rol\u00fcn\u00fcn \u00f6nemli dedekt\u00f6rleri olarak kullan\u0131labilir Beclin1, otofajinin iyi bilinen bir d\u00fczenleyicisidir ve otofaji ile pozitif korelasyon g\u00f6sterir.Beclin1, otofaji ve membran ta\u015f\u0131nmas\u0131 i\u015flevlerini yerine getirmek i\u00e7in VPS15, VPS34 ve ATG14 gibi proteinlerle etkile\u015fime girer.LC3, mayadaki ubikitin benzeri bir de\u011fi\u015ftirici olan ATG8'in bir homologudur LC3, mayadaki ubikitin benzeri de\u011fi\u015ftirici ATG8'in bir homologudur ve otofajide rol oynad\u0131\u011f\u0131 d\u00fc\u015f\u00fcn\u00fclmektedir. LC3, ATG4 ile muamele edildikten sonra C-terminal kal\u0131nt\u0131s\u0131n\u0131 kaybeder ve LC3-I'e d\u00f6n\u00fc\u015ft\u00fcr\u00fcl\u00fcr. LC3-I, otofagozom membran\u0131ndaki lipid molek\u00fcl\u00fc fosfatidiletanolamine kovalent olarak ba\u011flanarak bir dizi ubikitinasyon benzeri enzimatik reaksiyona u\u011frar ve LC3-II'ye d\u00f6n\u00fc\u015ft\u00fcr\u00fcl\u00fcr ve LC3-II'nin LC3-I'e oran\u0131ndaki bir art\u0131\u015f, y\u00fcksek otofaji seviyelerinin g\u00f6stergesidir. p62, otofajik aktiviteyi yans\u0131tan bir marker proteindir ve protein seviyeleri otofaji seviyesi ile ili\u015fkilidir. p62, otofaji aktivitesi i\u00e7in bir marker proteindir ve protein seviyesi otofaji ile negatif ili\u015fkilidir, \u00f6rn, otofaji ger\u00e7ekle\u015fti\u011finde, p62 proteini sitoplazmada par\u00e7alan\u0131r; otofaji aktivitesi zay\u0131flad\u0131\u011f\u0131nda ve otofaji i\u015flevi kusurlu oldu\u011funda, p62 proteini sitoplazmada birikecektir. Bu deneyde, NMN'nin Beclin1 ve LC3-II\/LC3-I proteinlerinin g\u00f6receli ekspresyonunu a\u015fa\u011f\u0131 reg\u00fcle edebilece\u011fi ve P-mTOR\/mTOR ve p62 proteinlerinin ekspresyonunu yukar\u0131 reg\u00fcle edebilece\u011fi Western blot ile do\u011frulanm\u0131\u015ft\u0131r. Ayr\u0131ca, NMN'nin OGD PC12 h\u00fccrelerinde otofajik vezik\u00fcllerin, otofajik lizozomlar\u0131n ve floresan noktalar\u0131n\u0131n say\u0131s\u0131n\u0131 ve yo\u011funlu\u011funu azaltabilece\u011fini do\u011frulamak i\u00e7in transmisyon elektron mikroskopisi ve MDC y\u00f6ntemi de uygulanm\u0131\u015ft\u0131r. Yukar\u0131dakiler, NMN'nin PC12 h\u00fccrelerinde OGD ile ind\u00fcklenen otofajiyi inhibe edebilece\u011fini g\u00f6stermektedir.
\nSonu\u00e7 olarak, belirli bir NMN dozunun PC12 h\u00fccrelerinde OGD kaynakl\u0131 otofaji hasar\u0131na kar\u015f\u0131 koyabilece\u011fi, b\u00f6ylece sitoprotektif bir etki g\u00f6sterebilece\u011fi ve bu koruyucu etkinin mTOR ile ili\u015fkili yolakla ili\u015fkili olabilece\u011fi \u00f6nerilmektedir. Bu \u00e7al\u0131\u015fma, OGD kaynakl\u0131 h\u00fccresel otofaji hasar\u0131n\u0131 \u00f6nlemek i\u00e7in NMN i\u00e7in belirli bir hedef referans\u0131 sa\u011flayabilir ve do\u011fal bile\u015fik NMN'nin geli\u015ftirilmesi i\u00e7in belirli laboratuvar verilerini biriktirebilir.<\/p>","protected":false},"excerpt":{"rendered":"

Kimyasal Ad\u0131: Beta-Nikotinamid Monon\u00fckleotid<\/p>\n

CAS No: 1094-61-7<\/p>\n

Molek\u00fcler Form\u00fcl: C11H15N2O8P<\/p>\n

Molek\u00fcl a\u011f\u0131rl\u0131\u011f\u0131:334.22<\/p>\n

G\u00f6r\u00fcn\u00fc\u015f: Beyaz toz<\/p>\n

Assay:97%~100.0%<\/p>","protected":false},"featured_media":0,"comment_status":"open","ping_status":"closed","template":"","meta":[],"product_brand":[],"product_cat":[113],"product_tag":[],"class_list":{"0":"post-6829","1":"product","2":"type-product","3":"status-publish","5":"product_cat-organic-intermediate","7":"first","8":"instock","9":"shipping-taxable","10":"product-type-simple"},"yoast_head":"\nChina beta-nicotinamide mononucleotide Manufacturer CAS 1094-61-7 - Longchang Chemical<\/title>\n<meta name=\"description\" content=\"beta-nicotinamide mononucleotide CAS 1094-61-7 Organic Intermediate is an organic intermediate for chemical synthesis and formulation manufacturing.\" \/>\n<meta name=\"robots\" content=\"index, follow, max-snippet:-1, max-image-preview:large, max-video-preview:-1\" \/>\n<link rel=\"canonical\" href=\"https:\/\/longchangchemical.com\/tr\/product\/beta-nicotinamide-mononucleotide-cas-1094-61-7\/\" \/>\n<meta property=\"og:locale\" content=\"tr_TR\" \/>\n<meta property=\"og:type\" content=\"article\" \/>\n<meta property=\"og:title\" content=\"China beta-nicotinamide mononucleotide Manufacturer CAS 1094-61-7 - 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